Providing resources and ideas for therapies and medical developments for Parkinson's disease:
Motivate the development of a new mercury chelator (because current offerings do more harm than good), for the purpose of mercury detoxification. With current mercury chelator options falling short, there is a compelling opportunity for pioneering chemists and research teams to lead an effort to develop a safer and more effective mercury detoxification process. Help shape a healthier world while unlocking immense potential for societal impact by championing this mission.
All humans are exposed to some level of mercury intoxication. The environment of planet Earth has become irreversibly polluted with the neurotoxin, mercury. The burning of coal releases ethyl-mercury into the atmosphere which distributes it worldwide. Mercury has entered the ecosystem of the oceans, poisoning fish which gets consumed by humans. Products such as Compact Fluorescent Light Bulbs (CFLs) when broken release mercury as do thermometers and thermostats which may employ mercury. The extraction of gold in "artisanal" small scale mines use and release an estimated 2000 tons of mercury into the environment annually (ref UN). The widespread use of silver-mercury amalgam dental fillings (estimated 91,000,000 in US in 2018 survey) has put a mercury leaching time-bomb into the mouths of many. Some recently published papers address the link between mercury intoxication and alpha-synuclein fibrillation and Parkinson's disease (reference: Mercury and Parkinson's Disease: Promising Leads, but Research Is Needed (Torrey and Simmons, 2023).
It is almost impossible to avoid mercury intoxication. Those coastal communities which rely on fish for food, those with dental amalgams or those exposed from any other factor are at risk of the neurological effects of mercury toxicity.
Hg is derived from the Latin name for mercury: hydrargyrum
Current chelator compounds are either ineffective at removing mercury from the body or they are ineffective at maintaining a bond with mercury resulting in the release of mercury allowing it to relocate to sensitive areas of the body such as the brain. A study by H. V. Aposhtian et al., found that current chelators (DMPS, DMSA, glutathione (GSH) and lipoic acid), alone or in combination, do not decrease the brain burden of mercury.
References:A study by the renowned researcher, Graham George, showed that current chelators such as DMPS or DMSA do not maintain a bond with mercury and do not bond in a fashion expected by chemists. DMPS and DMSA stir up mercury, where it then travels to high mercury affinity areas like the brain. Many of the chelators in use today were developed in the 1940's and 1950's and are innefective or outright dangerous.
Mercury chelation does not work!
References:Once the chelator has bonded with mercury, it has to be recognized by the kidneys or liver so that it may be evicted from the body as urine or as stool respectively. Not all chelators are effective at evicting mercury. This is true for the chelator NBMI which has been shown to fail at this task.
References:
For more discussion on mercury, chelation and Parkinson's see Mercury and Parkinson's Disease.
NBMI: this chelator bonds to the mercury atom (180 deg) but fails to be evicted from the human body
DMSA: this chelator does not typically bond in this geometric structure due to mercury's propensity for a 180 deg bond.
DMSA: Research by Graham George shows that the typical DMSA chelate structure with Hg is 2 DMSA bonding with 2 Hg. This chelator gets evicted from the body, primarily by the kidneys, but makes a structurally weak bond to the mercury atom and generally drops it before being evicted
Many of the chelation compounds in use today were developed during the WW2 era or at the very least, prior to the availability of tools such as "Computational Chemistry". These tools allow for the simulation of chemical reactions, compound optimization, compound discovery, virtual screening (VS) and high throughput screening (HTS). They are ideally suited for the discovery, simulation and optimization of potential chelators including the optimization and rework of current chelators. One key CADD feature is "docking" or the modeling of binding energy to a target where the bond of the chelator to mercury can be simulated. The virtual world allows for rapid development and experimentation.
Computer-Aided Drug Design (CADD) software:Compounds developed can be shared digitally in any of the following public databases:
While the virtual world of computational chemistry allows for rapid prototyping, at some point the chelator compound must be created in order to be tested or applied. There are many university and commercial labs that can perform this task.
A true validation to test if the chelator is not redistributing mercury to the brain, can only be performed on mice as an autopsy is required to measure the brain burden of mercury for both the mice testing the chelator and for the control specimens.
The following paper is a good example of chelator validation using mice: Vitamin C, Glutathione, or Lipoic Acid Did Not Decrease Brain or Kidney Mercury in Rats Exposed to Mercury Vapor (Aposhian et al., 2003)
This paper covers the mercury intoxication of mice, the measurement of the effects of chelation on the mouse brain and the effects of mercury mobilization.
The paper also covers compliance with animal treatment as well as the use of control and sentinal specimines.
There are some chelators which have not been shown to be qualified as safe from redistributing mercury and need to be validated. There are also some which have been improperly validated. Proof that a chelator is non-toxic, lowers the blood level of mercury or improves organ toxicity, is not a validation that the chelator does not increase the brain burden of mercury.
Chelators not tested or not adequately tested:
A new chelator could be used under "compassionate use" also known as "expanded access programs" (EAPs), "right to try", as a supplement which makes no claims, or get FDA phase 1, 2 and 3 approval.
This protocol relates to the use of an investigational medicine outside of a clinical trial to treat a serious or life-threatening condition. Mercury toxicity is often such a condition. A request for "Compassionate Use" or "Expanded Access" must be requested by a licensed doctor and approved by the FDA for an individual. Requests are made by phone or email. Adverse events must be immediately reported to the FDA.
EAPs conditions of use:See FDA expanded access contact information and forms.
A US law passed in 2018 provides another way to access unapproved drugs. Right to try is only for drugs in a FDA phase 1 trial. This law is used to request access to an unapproved drug from a company that makes it, without having to go through the FDA.
Consultants and contractors:
We have a chicken and egg problem. Those financing an endeavor want to see a research team who will develop the chelator. It's hard to put together a scientific team when we can't identify financing.
Potential Funding Sources:
This web page is meant to instigate and inspire a call to action to take a journey towards pioneering the development of a new mercury chelator. The demand it urgent. It is hoped that development teams can be formed to harness the power of modern computational chemistry tools to engineer a breakthrough solution for a safe and effective mercury chelation compound. This call to action is dependent upon the expertise and passion of dedicated scientists and research teams to bring this vision to life. Join us to unite and rally passionate scientists and experts to collaborate on the quest to safeguard global health and environmental well-being from the harmful effects of mercury.
Note that this website does not have the financial or technical means to undertake this call to action.
A listing on this web page does not mean that this website endorses any given business or institution.